D

Neostigmine benefits post-synaptic neurotoxicity (e.g., cobra) but is ineffective against the presynaptic β-bungarotoxin of kraits—clinical studies showed no improvement when anticholinesterase was tried in krait victims [Theakston, 1990?, PMID 2389328]. In contrast, polyvalent Indian antivenom does not neutralize Hypnale hypnale venom [Ariaratnam, 2008, PMID 18455743]; cobra venom is well known to be predominantly neurotoxic due to α-neurotoxins [Utkin, 2007, PMID 17979735]; and standard practice is to give atropine before neostigmine to block muscarinic effects during anticholinesterase therapy for elapid bites [Gold, 1996, PMID 8669746]. Therefore statement D is the only false option.