D

Hypoglycaemia in Von Gierke disease triggers catecholamine-mediated lipolysis, flooding the liver with fatty acids whose β-oxidation supplies acetyl-CoA for ketone production; at the same time, oxaloacetate is drawn into gluconeogenesis, limiting the TCA cycle and further diverting acetyl-CoA to ketogenesis [Blackett, 1979, PMID 6753728; Bach, 1978, PMID 87165]. Because fat mobilization is actually increased—not low—“low fat mobilization” cannot account for the ketosis, making option D the incorrect statement.